A nutrient chemical and pheromone-dependent neurogenic niche
The article linked below offers the first evidence I have seen for a diet-responsive neuogenic niche.
“Weight struggles? Blame new neurons in your hypothalamus.” May 21st, 2012.
Excerpt: “People typically think growing new neurons in the brain is a good thing – but it’s really just another way for the brain to modify behavior,” Blackshaw explains. He adds that hypothalamic neurogenesis is probably a mechanism that evolved to help wild animals survive and helped our ancestors do the same in the past.”
My comment:
In my model nutrient chemicals and pheromones have direct effects on hypothalamic neurogenesis, olfactory bulb neurogenesis, and hippocampal neurogenesis. These direct effects link nutrient chemicals and pheromones to the biological core of mammalian reproduction: the hypothalamic gonadotropin releasing hormone (GnRH) pulse.
In mammals, for example, food odors and pheromones cause changes in GnRH pulse frequency that result in patterns of neurogenesis responsible for the development of behaviors associated with proper food choice (individual survival) and proper mate choice (species survival). Food odors up-regulate and social odors down-regulate the calibration of stochastic gene expression responsible for control of speciation via hippocampal neurogenesis and the required learning and memory of species specific behaviors.
The direct effect of food odors and social odors on signalling pathways makes the epigenetic effects of chemical cues as important to the understanding of human behavior as they are to the understanding of behavior in every other species. This is especially true for placental mammals.
The in utero and postnatal effect of the chemical stimuli is on the development of the hypothalamic GnRH neuronal niche, which is responsible for the conditioning of food preferences and mate preferences and the transgenerational epigenetic inheritance of all behaviors required for individual and species survival. GnRH-dependent luteinizing hormone (LH) secretion, for example, is the link between proper nutrition and reproductive sexual behavior. The feedback loops are detailed in my model for the adaptive evolution of the human brain and behavior.
Kohl, J.V. (2012) Human pheromones and food odors: epigenetic influences on the socioaffective nature of evolved behaviors. Socioaffective Neuroscience & Psychology, 2: 17338.
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< 1 MIN READ
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Honeybees, food odors, and perfume
< 1 MIN READ
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Pheromones and a realistic model of sexual differentiation
3 MIN READ
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Sniffing out your nightlife / Pheromone-infused nightclubs
< 1 MIN READ
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Erox rises from the ashes
< 1 MIN READ
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Avian recognition of human faces
< 1 MIN READ
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Human Pheromones Affect Women's Behavior
2 MIN READ
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A lesser role for olfaction and pheromones in humans?
1 MIN READ
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Creating a Myth
< 1 MIN READ
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Study results: Human pheromones influence human behavior
2 MIN READ
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Social selection: genetic contribution of viruses to life on earth
< 1 MIN READ
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Natural scent of women linked to increased testosterone and seduction
< 1 MIN READ
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Too many good-smelling males is not good
< 1 MIN READ
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Pheromones and Physiology: Faculty of 1000: Naturally Selected
2 MIN READ
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Human pheromones in tears automagically effect testosterone
2 MIN READ
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Dialogue on pheromones: Avery Gilbert / James V. Kohl
2 MIN READ
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Mammalian pheromones are not a myth
3 MIN READ
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Molecular biology and social science theory
2 MIN READ
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Study Results: Human pheromones increase flirtatious behavior and ratings of attraction
2 MIN READ
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Results: increased flirtatious behavior and self-reported attraction
2 MIN READ
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Men perspire, women glow
< 1 MIN READ
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Food preferences and mate preferences
< 1 MIN READ
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Presentation on Human Pheromones delivered on August 3, 2010
2 MIN READ
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American and Canadian Mensa Presentation
< 1 MIN READ
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The Great Pheromone Myth (opinion/review)
19 MIN READ
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Welcome to Pheromones.com
< 1 MIN READ
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Scent of a woman: Men’s Testosterone Responses to Women's Scent
< 1 MIN READ
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Earlier evidence: pheromones influence testosterone in men
< 1 MIN READ
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