Olfactory perception of genetic information
(click to enlarge)
This open access journal article accurately represents what is currently known about nutrient-dependent RNA-directed DNA methylation and RNA-mediated events, which link the epigenetic landscape to the physical landscape of DNA via the de novo creation of olfactory receptors.
RNA-mediated amino acid substitutions stabilize thermodynamic cycles of protein biosynthesis and degradation, which enables the contribution of viral microRNAs to entropic elasticity unless nutrient uptake and nutrient-dependent microRNAs are linked to anti-entropic DNA repair via the physiology of reproduction. RNA-mediated amino acid substitutions differentiate all cell types of all individuals of all genera.
Reported as: Sniff tests used to create olfactory fingerprint
Excerpt:
…it would take just 10 odors and 11 descriptive words to accurately build an olfactory fingerprint for one individual, or taking it further, using 34 odors and 35 descriptive words to create olfactory fingerprints for everyone alive today.
See also: Olfactory Fingerprints
Excerpt:
Given the diversity of subjects’ responses, the researchers estimated that just 7 odors and 11 descriptors would have been sufficient to identify each of the 89 individuals based on their sense of smell. To individually identify any of the world’s 7 billion human inhabitants would require just 34 odors and 35 descriptors.
The idea that human biodiversity “evolved” to make the unique olfactory fingerprint of 7 billion people identifiable via the human sense of smell is an idea based on pseudoscientific nonsense.
In species from microbes to humans, pheromones control the physiology of reproduction, which is nutrient-dependent.
For examples, see: Nutrient-dependent/pheromone-controlled adaptive evolution: a model
See also: From Fertilization to Adult Sexual Behavior
Excerpt 1)
Data are available to suggest that postnatal environmental chemosensory behavioral imprinting may even influence human mate choice as occurs in mice. Among the Hutterian Brethren (the “Hutterites”), who live as somewhat social isolates, mating was nonrandom with respect to HLA haplotypes (Ober, Weitkamp, Elias, and Kostyu, 1993). Similarly, Wedekind et al. (1995) found that some genetically determined odor components can be important in human interactions.
Excerpt 2)
Kohl (1996) has proposed that the early prenatal development of the olfactory and VNO systems and of the GnRH neuronal system allow paranatal and postnatal exposure to pheromones the power to exert organizational and activational effects on behavior, whenever in life this exposure occurs. In so doing, these chemosensory stimuli link the social environment to basic genetically determined substrates of human behavior.
Empirical evidence of a link among human pheromones, olfaction, gene activation in GnRH neurosecretory neurons, neuroendocrinology, and behavior comes primarily from the study of other mammals. Nevertheless, the interaction between sensory input and neuroendocrinology appears to be a general rule in endocrine relationships that underlie behavior (LeMagnen, 1982).
See also: Sense of smell is strictly personal, study suggests
Excerpt:
This variation means that nearly every person’s sense of smell is subtly different.
My comment: This variation links nutrient-dependent RNA-mediated amino acid substitutions to cell type differentiation via metabolic networks and genetic networks in all genera via their biophysically constrained chemistry of protein folding.
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What I was most interested in were the researchers’ attempts to link the subjects’ olfactory components to components of their immune systems. Since other studies have provided details on how our immune systems become unique, it would follow that the subjects’ immune systems may have been the underlying cause for the study’s findings.
However, in the study’s limitations paragraph the researchers stated that this study didn’t prove such causes. Perhaps the causes for our “unique olfactory perception” will be researched in future studies.
https://surfaceyourrealself.com/2015/07/19/what-could-be-the-cause-of-humans-having-a-unique-sense-of-smell-surfaceyourrealself/
Mutations arise when the immune system is overwhelmed. Typically,
nutrient-dependent microRNAs keep the proliferation of viral microRNAs
from causing irreparable damage to DNA. The nutrient-dependent microRNAs
support immune system function via the de novo creation of olfactory
receptor genes.
See also: https://rna-mediated.com/anti-entropic-containment-of-energy-symbiosis-1-0/
and https://rna-mediated.com/new-comments-posted-on-science-x-network/
“Mechanisms of stress in the brain”
and other published works by serious scientists who understand that you
must start with the epigenetic landscape and link it to the physical
landscape of DNA. You must link what is known about physics, chemistry,
and conserved molecular mechanisms from atoms to ecosystems, both before
and after the mechanisms are perturbed by viruses. Viruses cause
mutations.