Zika infection may affect adult brain cells

Illumination of the fluorescent biomarker in green revealed that the adult mouse brain could be infected by Zika in a region called the subgranular zone of the hippocampus. Full of neural progenitor cells, this part of the brain is important in learning and memory. Credit: Laboratory of Pediatric Brain Disease at The Rockefeller University/Cell Stem Cell

As reported by Bob Yirka in: Best of Last Year – The top Medical Xpress articles of 2016

a team of MIT biologists reported that amino acids, not sugar, supplied most of the building blocks for tumor cells—cell mass was not mostly glucose, as they had expected.

In other news, at team at Duke University found a key protein for spinal cord repair

Most of the pseudoscientific nonsense reported in the top Medical Xpress articles from the past can be compared to the facts about how energy-dependent viral latency is required for ecological adaptation in the context of RNA-mediated amino acid substitutions.

The facts were reported as “New CRISPR–Cas systems from uncultivated microbes

See also: More refutations of neo-Darwinian nonsense and Dietary lutein and pheromone-controlled brain development

All serious scientists have learned there is no “new CRISPR–Cas” system. The de novo creation of the innate immune system links nutrient energy-dependent changes from angstroms to ecosystems via hydrogen-atom transfer in DNA base pairs in solution. The energy-dependent changes in base pairs are the obvious links to the development of morphological and behavioral phenotypes in all living genera. All phenotypic expression is energy-dependent, and it must link autophagy to the physiology of reproduction. For example, in species from microbes to humans Feedback loops link odor and pheromone signaling with reproduction

The pheromone-controlled physiology of reproduction links the energy-dependent creation of supercoiled DNA to protection from virus-driven energy theft and all pathology. Simply put, supercoiled DNA prevents the genomic entropy that is linked to all pathology.

For comparison, the link from the anti-entropic virucidal energy of sunlight to supercoiled DNA via the viral hecatomb in archaea is the link to the transgenerational epigenetic inheritance of Zika virus-damaged DNA in human infants. Facts about energy-dependent polycombic ecological adaptations will put an end to ridiculous theories about mutations and evolution. That will become perfectly clear if Gunter Witzany allows others to help present  what is known about energy-dependent changes in the microRNA/messenger RNA balance at the 2018 conference he reportedly is organizing.

See: Royal Society: The Public Evolution Summit (p. 12)

 … evolution of genome invading RNA networks that edit host g… Witzany is organizing a conference symposium for July 2018 “Evolution-genetic innovations without error replication”

Until everyone learns how energy-dependent autophagy and supercoiled DNA prevent error replication, pseudoscientists may continue to tout their ridiculous theories. Many more people will suffer and die prematurely due to biased reporting that supports the evolution industry and “big bang” cosmology industry.

But wait, 2016 is not over yet, is it?

See: Spatiotemporal antagonism in mesenchymal-epithelial signaling in sweat versus hair fate decision

This was reported by Bob Yirka as: Researchers identify signals during embryonic development that control the fate of skin cells to be sweaty or hairy

See also: Royal Society’s Sir Venki Ramakrishnan Agrees to Post Public Evo Audio

…public discussion from the recent “new trends” in evolution conference will be posted shortly online on the Royal Society event webpage.

The public discussion may help to reveal that the fate of all cell types is energy-dependent and RNA-mediated via natural selection for codon optimality linked to autophagy and to supercoiled DNA.

See for example: rs3827760, also known as 1540T/C, 370A or Val370Ala, is a SNP in the ectodysplasin A receptor EDAR gene on chromosome 2.

This SNP and everything known about how chromosomal rearrangements are linked to energy-dependent  pheromone-controlled biodiversity, which links the mouse model of cell type differentiation to humans via what is known about energy-dependent RNA-mediated amino acid substitutions such as Val370Ala. The facts about the Val370Ala and other amino acid substitutions have been known since 1973 and four more decades of facts were detailed in the context of this 2013 review.

See for other examples: Nutrient-dependent/pheromone-controlled adaptive evolution: a model

These two reports (Grossman et al., 2013; Kamberov et al., 2013) tell a new short story of adaptive evolution. The story begins with what was probably a nutrient-dependent variant allele that arose in central China approximately 30,000 years ago. The effect of the allele is adaptive and it is manifested in the context of an effect on sweat, skin, hair, and teeth. In other mammals, like the mouse, the effect on sweat, skin, hair, and teeth is due to an epigenetic effect of nutrients on hormones responsible for the tweaking of immense gene networks that metabolize nutrients to pheromones. The pheromones control the nutrient-dependent hormone-dependent organization and activation of reproductive sexual behavior in mammals such as mice and humans, but also in invertebrates as previously indicated. That means the adaptive evolution of the human population, which is detailed in these two reports, is also likely to be nutrient-dependent and pheromone-controlled, since there is no other model for that.

Each week’s science news brings additional representations of facts that refute all neo-Darwinian pseudoscientific nonsense by linking Darwin’s “conditions of life” to all biodiversity. Most science news outlets still report and/or tout pseudoscientific nonsense because the reporters missed the fact that life is nutrient-dependent and the physiology of reproduction is controlled by pheromones in species from microbes to humans.

Can you imagine how embarrassed you would be if you had not learned to include the fact that all organisms must eat or they cannot reproduce in your reports. That fact means organisms cannot become part of a species if they can’t find food. What’s worst about having to explain that fact to reporters and/or pseudoscientists is that no experimental evidence of biologically-based cause and effect supports claims that one species ever evolved into another.

“The Battlefield”

I may have mentioned this before.  I’ve been banned from participation on this group, twice. Most recently, I complained that two other admins support Peter Berean’s attempts to denigrate my life’s works on energy as information-dependent changes in the microRNA/messenger RNA balance and denigrate me personally with his claims about “bio-functional information.”

By default, Larry Kisner Sr., and John L Leonard support the ignorant representations made by Peter Berean, and there is rarely any accurate representation of energy-dependent biologically-based cause and effect.

I’ve suggested several times that the group may be another “False Flag” group. Others can evaluate the claims made there for comparisons to the accurate claims of serious scientists who have linked hydrogen-atom transfer in DNA base pairs in solution to energy-dependent changes in microRNA flanking sequences and all biodiversity via natural selection for energy-as-information-dependent codon optimality.

You’ll be learning more about the facts as others try to obfuscate them in the weeks to come. Until then, see this conclusion from my 2013 review:

…the model represented here is consistent with what is known about the epigenetic effects of ecologically important nutrients and pheromones on the adaptively evolved behavior of species from microbes to man. Minimally, this model can be compared to any other factual representations of epigenesis and epistasis for determination of the best scientific ‘fit’.


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