Achromobacter and killer theories

By: James V. Kohl | Published on: November 14, 2017

A pan-genomic approach to understand the basis of host adaptation in Achromobacter 

Ecological variation is linked to food energy-dependent biophysically constrained viral latency by the pheromone-controlled physiology of reproduction in species from microbes to humans.

For comparison, these authors claim:

This study provides strong phylogenetic and pan-genomic bases to motivate further research on Achromobacter, and contributes to the understanding of opportunistic pathogen evolution.

Their claim exemplifies how teaching neo-Darwinian theory causes unnecessary suffering and how it may cause the premature death of our loved ones. Physicians who do not know the difference between the human idiocy of claims about pathogen evolution and the facts about pathogen adaptation to a host are not likely to ever link the conserved molecular mechanisms of energy-dependent RNA-mediated cell type differentiation to healthy longevity.

That means they are not likely to link the virus-driven degradation of messenger RNA from mutations to all pathology. They will never look for the results of a search like this:

bacteriophage achromobacter For example: (1974) Bacteriophages and cryptic lysogeny in Achromobacter

See this explanation of energy-dependent viral latency: Viral replication: lytic vs lysogenic (video 5:10) Instead of accurately representing the anti-entropic virucidal effect of UV light, as explained here: Energy as information and constrained endogenous RNA interference (video 6:46)

How much longer will it be until everyone realizes that teaching students to believe in mutation-driven evolution leads them to become professors who are the biologically uninformed science idiots? The professors teach medical professionals to believe in the “bad luck” theories of pathology. See for comparison to the “Bad Luck” theory of random mutations.

In the world of serious scientists, bacteria become archaea and humans become non-human primates in the context of the viral hecatomb (i.e., the virus-driven degradation of messenger RNA). In the world of pseudoscientists who teach physicians to believe in “bad luck” theories, the only hope that most people have is the fact that energy-dependent polycombic ecological adaptations typically biophysically constrain viral latency.

Clearly, bacteriophages (viruses in bacteria) can steal quantized energy to proliferate and kill. Evolutionary theorists are willing to let that continue to happen. Are you?

If not, see: Cytosis

See also: To save a young woman besieged by superbugs, scientists hunt a killer virus

Part of the issue is that phages [viruses] are alive, and continue evolving inside the body.

Viruses are not “alive” and nothing alive evolves inside or outside the body. Organisms adapt to the virus-driven degradation of messenger RNA, or species become extinct.

See also: Episode 10: A giant Petri dish chock-full of superbugs shows evolution as it happens  by Carl Zimmer

…you can observe the evolution of antibiotic resistance in a series of bursting waves. It’s so eminently viewable, in fact, that the MEGA plate video has been watched almost 25 million times since it was posted online in September.

The pseudoscientific nonsense touted by journalists like Carl Zimmer will continue to cause unnecessary suffering and premature deaths. The pathology will be attributed to antibiotic resistance, instead of the failure to teach intelligent students about food energy-dependent pheromone-controlled ecological adaptations.

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