vs Nature Publications Group

By: James V. Kohl | Published on: December 14, 2017

Autophagy is the antiphage defense strategy (James V. Kohl: December 8, 2016) was launched on 11 December 2017

See for comparison from the Nature Publications Group.  First, they try to establish a more complicated new definition with several different pathways


Autophagy is a process by which cellular material is degraded by lysosomes or vacuoles and recycled. Several autophagy pathways operate within a cell, including macroautophagy, microautophagy and chaperone-mediated autophagy.

After the new definition, and the extension of vague processes and magical pathways to macroautophagy, microautophagy and chaperone-mediated autophagy, we see:

Latest Research and Reviews

An and Harper quantify ribophagy in mammalian cells and show that nutrient-deprivation-induced ribophagy is independent of the ATG8 conjugation system, whereas proteotoxic stress-induced ribophagy requires ATG5 and VPS34.

The similarities and differences between the energy-dependent changes in epigenetic effects of nutrient stress and the epigenetic effects of social stress has not been addressed in published works until recently. But see: microRNA Items: 1 to 20 of 67725.
See for comparison: ribophagy Items: 1 to 20 of 29
Food energy-dependent changes in the microRNA/messenger RNA balance biophysically constrain viral latency in species from microbes to humans. The virus-driven degradation of messenger RNA has been linked to all pathology. The “Nature Publications Group” tries to sneak up from behind with the less detailed link from ribophagy to autophagy.
Research | 11 December 2017
Systematic analysis of ribophagy in human cells reveals bystander flux during selective autophagy

Research | 11 December 2017 | open
SPLICS: a split green fluorescent protein-based contact site sensor for narrow and wide heterotypic organelle juxtaposition
Research | 11 December 2017
Autophagy induced during apoptosis degrades mitochondria and inhibits type I interferon secretion
Research | 11 December 2017 | open
RAB37 interacts directly with ATG5 and promotes autophagosome formation via regulating ATG5-12-16 complex assembly
Sour grapes?

Nobody wants to belong to the party of losers. One of the best strategies in such a case is evidently an interpretation of the change as a gradual accumulation of knowledge while their work has always been at the cutting edge. — Kalevi Kull

The losers at the “Nature Publications Group” banned me from commenting on any article (~two years ago). Predictably, the “Nature Publications Group” and all others will be forced to link the virus-driven degradation of messenger RNA to all pathology and eliminate the pseudoscientific nonsense touted by neo-Darwinian theorists and “Big Bang” cosmologists.
See also:  Journal of Genetics and DNA Research Articles in Press | Volume 1, Issue 1
The Gene Polymorphism of VMAT2 Decrease the Risk of Developing SCZ in Male Han Chinese
Biophysical and Biochemical Mechanisms of Forming and Development A Human Eukaryotic Organism from Single Pluripotent Cell into Multicellular Embryo and A Living Organism in Norm
Mouse Models for NASH: Are we there yet?
Are we All Transgenic?
Genetic Engineering 2020
On Novel Anti-viral HIV Gene Editing Platforms
2016 – 2017 Historical context:
MicroRNAs and Autophagy: Fine Players in the Control of Chondrocyte Homeostatic Activities in Osteoarthritis
Emerging connections between RNA and autophagy
I founded the domains (1995), (2015) and (2017) because the food energy-dependent species-specific production of pheromones clearly links feedback loops to all biophysically constrained RNA-mediated biodiversity via autophagy.

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