Environmental selection is natural selection (3)
Friends and family members have already seen the effects of the molecular mechanisms in the changes in plant growth in the channels of the lakes, and elsewhere. Prevention of the changes is always better than treatments after the uncontrolled growth has occurred.
One of the main mechanisms of persistent NF-κB activation in KSHV-infected cells is dysregulation of host gene expression and their normal functions by viral oncogenes.
Taken together, these findings suggest that CADM1 plays a key role in the survival and growth of cancer cells associated with KSHV infection.
This is especially notable because CADM1 is known to play the opposite role in other cancers, suppressing the growth of melanoma, lung cancer, and other solid tumors. According to the authors, “targeting human protein CADM1 in viral-mediated cancers would be ultimate therapy against KSHV and HTLV-I associated malignancies.”
The virus-driven degradation of messenger RNA was linked to cancer but quantized energy/food energy-dependent prevention was not linked to fixation of the RNA-mediated amino acid substitutions that biophysically constrain viral latency. They claim that the opposite roles are especially notable. I agree. They can and should be used as example of human idiocy.
The microRNA-mediated link from algae blooms to cancer prevention and effective treatment is perfectly clear to all serious scientists. But there is an army of biologically uninformed teachers who refuse to learn how the creation of the sun is linked to everything else. They teach pseudoscientific nonsense about mutations and evolution to children, instead.
For example: Environmental selection during the last ice age on the mother-to-infant transmission of vitamin D and fatty acids through breast milk
Reported as: Gene linked to breastfeeding may have boosted survival of earliest Americans
“Teeth telling us something about fertility? That’s really amazing,” says biological anthropologist Julienne Rutherford of the University of Illinois in Chicago, who was not involved with the work.
See for comparison: System-wide Rewiring Underlies Behavioral Differences in Predatory and Bacterial-Feeding Nematodes (2013)
1) Predation in diplogastrid nematodes such as P. pacificus is a behavioral mode discrete from bacterial feeding and represents an evolutionary novelty unknown from C. elegans or their most recent common ancestor.
2) Pristionchus and other diplogastrid nematodes carry living bacteria in the gut, and they gain energy from organismic interactions during bacterial feeding (Rae et al., 2008). Together, the extended feeding repertoire of P. pacificus has resulted in a gain of structures in the anterior and in a loss in the posterior pharynx.
“The patterns of synaptic connections perfectly mirror the fundamental differences in the feeding behaviours of P. pacificus and C. elegans”, Ralf Sommer concludes.
P. pacificus is a predatory nematode with teeth. The synaptic connections linked from the feeding behaviors to the teeth are examples of nutrient energy-dependent pheromone-controlled ecological adaptations in the context of environmental selection.
See also: Virus-mediated archaeal hecatomb in the deep seafloor
Reported as: Deep-Sea Viruses Destroy Archaea
The facts about the virus-driven degradation of messenger RNA continue to be misrepresented in virtually everything that is tailored to the touting of pseudoscientific nonsense linked to ridiculous theories.
See also: Global warming transforms coral reef assemblages
“When corals bleach from a heatwave, they can either survive and regain their color slowly as the temperature drops, or they can die.
The virus-driven degradation of messenger RNA causes the coral bleaching. The heat stress facilitates viral replication, which is controlled by thermodynamic cycles of microRNA biogenesis.
Coral Reef Experiment Shows That Acidification from Carbon Dioxide Slows Growth
Ocean acidification will severely impair coral reef growth before the end of the century if carbon dioxide emissions continue unchecked
Acidification promotes viral replication at the level of hydrogen-atom transfer in DNA base pairs in solution.