From microRNA.pro to quantumsouls.pro (3)

Naturally occurring RNA interference has been patented as RNA-Guided Human Genome Engineering. Biophysically constrained viral latency at the level of quantum physics (energy-dependent changes in base pairs) is the link to sympatric speciation. That fact is exemplified by the diet-driven changes from C. elegans to the predatory nematode P. pacificus. The virus-driven degradation of messenger RNA forces diet-driven adaptations in P. pacificus, which include “teeth.” Changes in behavior are linked to its acquired ability to eat other nematodes.

I’ve had co-workers that I knew would adapt their behavior so that they could eat me if they got hungry enough. Congresswoman Alexandria Ocasio-Cortez will probably use the nematode model from her 2007 award-winning poster session to show that the nonsense about gene-editing was nothing more than another money making scam that paid well or fed everyone involved, mostly other Democrats in the USA.

She will force the change to less expensive and more effective treatments with RNA interference in the context of what has been referred to as a “billion dollar baby,” Get Well in the RNAi Way-RNAi, A Billion Dollar Baby in Therapy

Mitochondrion (aka John Hewitt) wrote:

Might this platform or something similar be used to edit mitochondria?
Could one make use of phase specific information, strand differences or SNPs at specific locations so that only one chromosome is targeted, presumable an offending heterozygous allele?

jvkohl  Food energy is something similar. It is already used to naturally edit mitochondria in the context of natural selection for energy-dependent codon optimality via RNA interference. The creation of ATP links the creation of RNA from RNA interference to fixation of amino acid substitutions and supercoiled DNA via the physiology of reproduction, which biophysically constrains viral latency. The pathways have been detailed at every level of examination from subatomic particles and cytosis to autophagy and healthy longevity.
Mitochondrion (aka John Hewitt) wrote:

I would have put the brakes on there at the end of sentence number 2 my man, or at least at supercoiled, and definitely by latency.

I’ve dealt with his level of ignorance before and he knows I do not care where he would put the brakes on. I think he wants me to put the brakes on so that he can start moving again with a sudden energy jump that links mutations to evolution. So, I wrote:
jvkohl 

jvkohl 

John Hewitt (Mitochondrion) and others like him want to prevent others from learning the facts about ecological adaptations.

See for comparison: Identification of evolutionarily conserved gene networks mediating neurodegenerative dementia (12/3/18) and HHV-6 encoded small non-coding RNAs define an intermediate and early stage in viral reactivation (9/5/18), which was reported as: Viruses under the microscope https://www.rdmag.com/news/…

“The virus hides in the genomic DNA”

Energy-dependent biophysical constraints on supercoiled DNA typically ensure viral latency by preventing stress-linked viral activation. Facts about human endogenous retroviruses (HERVs) have been linked to the viruses that supposedly hide in the organized genomes of other species for more than 2 decades. Reports that claim viruses hide are anthropocentric and gene-centric. They are among the most foolish of all reports published by biologically uninformed theorists and reported by so-called “science journalists.”

I do not expect any more discussion about the fact that CRISPR is Going to be Replaced. Few people seem to know enough about biophysically constrained cell type differentiation to understand that it is energy-dependent. By starting with mutations (de Vries, sudden energy jumps) they miss the facts about the virus-driven degradation of messenger RNA that links mutations to all pathology.
Having been caught touting ridiculous theories that still are based on a definition from the turn of the 20th century, people like John Hewitt must duck and run for cover, as serious scientists continue to detail cause and effect.
See for example, A viral Sm-class RNA base-pairs with mRNAs and recruits microRNAs to inhibit apoptosis (2017)

Our results uncover a role for this viral Sm-class RNA as a microRNA adaptor in the regulation of gene expression that follows precursor mRNA processing.