Trump-hating advocates of vaccines + GMOs (3)
MicroRNA.pro: Trump-hating advocates of vaccines + GMOs (1)
RNA-mediated.com: Trump-hating advocates of vaccines + GMOs (2)
microRNA + autophagy See items 1-1204
Scientific creationists from the Institute for Creation Research (ICR) and biologically uninformed neo-Darwinian theorists from Oded Rechavi’s group in Israel have helped Trump-hating advocates of vaccines + GMOs in the same way.
They have plagiarized my life’s works and ignored the facts about light-activated biophysical constraints on the assembly of the microRNA-RNA-peptide nanocomplex. The biophysical constraints link epigenetic effects on viral latency from the creation of sunlight to the physiology of reproduction and ecological adaptations. The epigenetic effects vary with the amount of energy as information that comes from sunlight, which links the energy to the type of food that insects and mammals eat. The metabolism of food to pheromones links feedback loops from the physiology of reproduction to autophagy and healthy longevity. Healthy longevity exemplifies the ecological adaptations.
Nutrient-dependent/pheromone-controlled adaptive evolution: a model (2013)
Nutrient-dependent Pheromone-Controlled Ecological Adaptations: From Angstroms to Ecosystems (2018)
ICR portrays part of the systems complexity of biologically-based ecological adaptations in their series on Engineered Adaptability:
See: Biological Networks Feature Finest Engineering Principles (January 2019) and New Distributed Problem-Solving Model for Population Adaptation (April 2019)
…most adaptations—broadly mischaracterized as “evolution” by secular science—are highly regulated, often rapid, and so clearly targeted to specific conditions that they are predictable.
Unfortunately, their researchers limited scientific progress with conflicting claims about viruses on several occasions.
Did God Make Pathogenic Viruses? (1999)
Pathogenesis is evidence of something gone wrong, a mutation or the accidental movement of genes, and not evidence of a system deliberately designed to cause human disease and suffering.
…viruses are not living, and in order to reproduce and to make ATP, they require all of the complex cellular machinery present in bacterial cells. Other scientists speculate that a reverse symbiosis occurred, and that viruses arose out of cell parts such as bacterial plasmids and other organelles, and eventually evolved into separate forms of life.
If viruses are not living, why would the claim about their need for ATP be linked to the evolution of separate forms of life?
The plot thickens: Viruses–Architects of the Brain? (2015)
Surely the Creator God of the Bible would be a better candidate than viruses for orchestrating the coordinated and integrated tasks that TEs perform in constructing mammal brains.
And then, suddenly: Celebrate Your Inner Virus (2017)
It is important that we understand the design present in viruses because God made them. All creatures of our God demonstrate his handiwork, and viruses are no different.
In two decades, the question about God’s creation of viral pathogens went from His Creation of all energy-dependent biodiversity on Earth to His Creation of viruses that have been linked from the degradation of messenger RNA to mutations and all pathology.
See for comparison:
Who rules the waves? – Viruses might just be bit players in the drama of the seas. Then again, they could be major actors (1996)
Most consider viruses to be a legion of cripples, sterilised by ultraviolet radiation and rendered impotent by hosts that are largely immune to their threat. But a few researchers take the opposite view. And if they turn out to be right, viruses could radically alter the balance of life in the oceans, ripping away huge parts of the food web that supports whales, sea birds and the fisheries on which many people rely.
To reduce the amount of confusion introduced by those who promote evolution in the context of the medical-industrial complex for comparison to those who seem to support scientific creationism, see:
Eukaryotic plankton diversity in the sunlit ocean (2015)
Virus-mediated archaeal hecatomb in the deep seafloor (2016)
See also: From Fertilization to Adult Sexual Behavior (1996)
Small intranuclear proteins also participate in generating alternative splicing techniques of pre-mRNA and, by this mechanism, contribute to sexual differentiation in at least two species, Drosophila melanogaster and Caenorhabditis elegans…
The term pre-mRNAs became the term microRNAs in more that 86,000 indexed published works before this extension of our claims from 1996 and my model Nutrient-dependent/pheromone-controlled adaptive evolution: a model was reported in:
Pheromones and Nutritional Signals Regulate the Developmental Reliance on let-7 Family MicroRNAs in C. elegans (5/16/19)
…pheromones correct heterochronic developmental cell lineage defects…
For comparison, see what happens in the context of gene editing or use of viruses in vaccines when no corrections occur:
Off-target Effects in CRISPR-Cas9 Genome Editing: Securing Specificity (1/24/19)
“Most investigation of off-target base editing has focused on DNA, but we have found that this technology can induce large numbers of RNA alterations as well,” study coauthor J. Keith Joung of Massachusetts General Hospital and Harvard Medical School says in a statement. “This surprising finding suggests the need to look at more than just genetic alterations when considering unintended off-target effects of base editors in cells.”
Revealing the determinants of codon usage bias is central to the understanding of factors governing viral evolution. Herein, we report the results of a survey of codon usage bias in a wide range of genetically and ecologically diverse human RNA viruses. This analysis showed that the overall extent of codon usage bias in RNA viruses is low and that there is little variation in bias between genes. Furthermore, the strong correlation between base and dinucleotide composition and codon usage bias suggested that mutation pressure rather than natural (translational) selection is the most important determinant of the codon bias observed. However, we also detected correlations between codon usage bias and some characteristics of viral genome structure and ecology, with increased bias in segmented and aerosol-transmitted viruses and decreased bias in vector-borne viruses. This suggests that translational selection may also have some influence in shaping codon usage bias.
The facts about microRNA-mediated cell type differentiation that link our claims from 1996 about C. elegans and other organisms to all nutrient-dependent pheromone-controlled biodiversity on Earth will be reestablished in Genome-Wide Study Reveals a Novel Regulatory Pathway: Translation Affects mRNA Stability in a Codon-Dependent Manner 6/26/19
All serious scientists know that this will be the end of all pseudoscientific nonsense touted by neo-Darwinian theorists and Big Bang cosmologists because Bazzini et al, published
Codon identity regulates mRNA stability and translation efficiency during the maternal-to-zygotic transition (2016)
While we observe a correlation between tRNA levels and the extremes of optimality, it is likely that this code integrates multiple inputs of translation from tRNA availability and accuracy (Akashi, 1994; Ishimura et al, 2014; Hussmann et al, 2015), tRNA modification (Gustilo et al, 2008; Novoa et al, 2012), to peptide bond formation, amino acid identity, and other factors influencing the translocation rate and ultimately elongation (Baragana et al, 2015; Faller et al, 2015). Future studies will be needed to determine how the ribosome or accessory proteins decode codon optimality (Richter & Coller, 2015), and how this code is regulated across cellular transitions, tissues, and pathological states.
The obvious link from the creation of the sun’s anti-entropic virucidal energy to the light-activated assembly of the microRNA-RNA-peptide nanocomplex and amino acid identity ensures the ridicule of all biologically uninformed theorists and all proponents of vaccines and all proponents of GMOs.