Pheromones protect us from viruses (4)
Have you tried and repeatedly failed to put the pieces of a puzzle together? Did you claim it was impossible for anyone else to do it? To me, that exemplifies atheism.
This helps to define atheism.
…a broad signalling cascade mediated by changes in phosphorylation, gene expression and vesicular trafficking that result in increased nutrient utilisation and storage, and suppression of catabolic processes.
The attempt to define the underlying defect can be compared to fact about the defect. See: Dependence of RNA synthesis in isolated thymus nuclei on glycolysis, oxidative carbohydrate catabolism and a type of “oxidative phosphorylation”(1964)
The synthesis of RNA in isolated thymus nuclei is ATP dependent.
You can link the ATP-dependent synthesis of RNA from insulin action in type 2 diabetes to Visualizing a protonated RNA state that modulates microRNA-21 maturation 10/26/20 via the 11/23/19 preprint.
The preprint was, in part, the basis for former President Donald J. Trump’s claim that sunlight and humidity help to prevent the replication of coronaviruses. 4/23/20
Since then, all intelligent serious scientists have continued to link the definition of atheism disbelief or lack of belief in the existence of God or gods to the overwhelming ignorance and arrogance of stupid theorists who failed to link RNA interference from prevention of type 2 diabetes to biophysical constraints on SARS-CoV-2.
For comparison, I claim that the light-activated assembly of the microRNA-RNA-peptide nanocomplex is the key to solving the puzzle of biophysically constrained viral latency and healthy longevity. My claim is supported by more than 117,564 indexed published works. See: pubmed.ncbi.nlm.nih.gov/?term=microRNA
It trains computational biologists, other atheists, and all fools to recognize light-activated top-down causation for prevention of virus-driven diseases.
Their “variational autoencoder” is energy-dependent. The variations are light-activated in the context of the assembly of the microRNA-RNA-peptide nanocomplex, which biophysically constrains viral latency via protonated RNA interference.
See: Visualizing a protonated RNA state that modulates microRNA-21 maturation 10/26/20 or the 11/23/19 preprint. The preprint was, in part, the basis for former President Donald J. Trump’s claim that sunlight and humidity help to prevent the replication of coronaviruses. 4/23/20
I claim that the light-activated assembly of the microRNA-RNA-peptide nanocomplex is the key to solving the puzzle of biophysically constrained viral latency and healthy longevity. My claim is supported by more than 117,350 indexed published works. See: pubmed.ncbi.nlm.nih.gov/?term=microRNA
See also my “roots to shoots” series. This is the latest open access refutation of stupid theories:
The authors refute the “Antagonistic Pleiotropy Theory” for the evolution of aging and replace it with my model of Nutrient-dependent/pheromone-controlled adaptive evolution 6/14/13, albeit indirectly and without attribution.
Clearly, however, the epigenetic effects of food odors and pheromones are involved in neurogenic niche construction as exemplified in nematodes (Bumbarger, Riebesell, Rödelsperger, & Sommer, 2013), and in flies (Swarup et al., 2013).
Bumbarger et al. (2013) identified:
Massive neural circuit rewiring between nematodes with divergent feeding behavior.
The divergent feeding behavior was linked from the pheromone-regulated genetic processes of reproduction to sympatric speciation in predatory nematodes with “teeth” and in flies via Thomas Hunt Morgan’s works in 1910 –and the forthcoming STEM toy for ages 14+. Genotype
This report on Bumbarger et al., (2013) links The neurobiological consequence of predating or grazing to a claim about evolution that supposedly occurred across 200 million years in two worm species.
Cori Bargmann, who studies the influence of genetics and the environment on the nervous system won 3 million for failing to link what nematodes eat to the physiology of food energy-dependent pheromone-regulated genetic processes and predatory sympatric speciation during the past 5-6,000 years of ecological adaptations.
See for comparison:
The findings confirm their earlier work suggesting that the majority of variants, including potentially harmful ones, were picked up during the past 5,000–10,000 years.
If predatory human behavior arose during the same time that predatory behavior in nematodes arose, no experimental evidence of top-down energy-dependent causation could be found to support stupid neo-Darwinian theories.
Instead, the claims from Physiology is rocking the foundations of evolutionary biology 4/12/13 would continue to make theorists look even more foolish.
Perhaps the elegant mathematics and the extraordinary reputation of the scientists involved blinded us to what now seems obvious: the organism should never have been relegated to the role of mere carrier of its genes.