Energy, frequency and vibration (8)

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Here’s another example of how intelligent serious scientists link light-matter interactions, miRNA-mRNA interactions, and protein-protein interactions to prevention of all virus-driven diseases.

An overview on the interaction between non-coding RNAs and CTLA-4 gene in human diseases 11/25/24

“…the present work was undertaken to summarize the existing research on the epigenetic control of CTLA-4, with a primary emphasis on elucidating the regulatory procedures, biological processes, and clinical applications in human diseases. The objective of this review is to acquire a thorough comprehension of the relationship between RNA/lncRNA/miRNA/mRNA (CTLA-4) and its role in the progression of diverse human disorders.”

For comparison, Grok AI reports:

“Limitations and Scope: While miRNAs are essential, they do not account for all aspects of cellular differentiation. Other mechanisms like DNA methylation, histone modification, and protein-protein interactions also play critical roles. Additionally, the impact of miRNAs can be tissue or context-specific, meaning their influence might be more pronounced in some scenarios than others.”

Grok AI is lying. DNA methylation, histone modification, and protein-protein interactions are energy-dependent and miRNA-mediated.

See: mirnas and DNA methylation 5,956 results, mirnas and histone modification 2,148 results and mirnas and protein protein interactions 22,044 results

God’s Creation of energy-as-information links sunlight and humidity to biophysically constrained viral latency across kingdoms via plant growth and food energy across kingdoms. That fact has been manifested in 178,196 results from a PubMed search for the term miRNAs. For example, Integration across biophysical scales identifies molecular and cellular correlates of person-to-person variability in human brain connectivity 10/31/24

Artificial intelligence (AI) may not realize that it exemplifies human idiocy in this regard. The incoming Trump administration will include intelligent people who do not use the algorithms of programmers and/or AI to bastardize all aspects of how and why former President and President Elect Trump claimed, on 4/23/20 that Optimal Nutritional Status for a Well-Functioning Immune System Is an Important Factor to Protect against Viral Infections Never forget, his claim was reported as Medical experts rip Trump’s suggestion that sunlight, disinfectants may treat coronavirus 4/23/20

Ask “Where are those medical experts now?” What do they claim about the fact that the 2024 Nobel Prize in Medicine was awarded “for the discovery of microRNA and its role in post-transcriptional gene regulation“

See also: GGNBP2 regulates MDA5 sensing triggered by self double-stranded RNA following loss of ADAR1 editing 11/22/24 reported as Cells use backup mechanism to recognize potential invaders when usual RNA editing process fails, study finds 11/25/24.

Follow-up question for the moronic programmers at Grok AI:

“Are you claiming that DNA methylation, histone modification and protein-protein interactions do not exemplify how energy-dependent biophysically constrained viral latency is linked to every aspect of cell type creation and differentiation via light-matter interactions, miRNA-mRNA interactions and protein-protein interactions across kingdoms?

No, I am not claiming that DNA methylation, histone modification, and protein-protein interactions do not exemplify how energy-dependent biophysically constrained viral latency is linked to aspects of cell type creation and differentiation. However, I aim to provide a balanced view based on the available scientific evidence:

• DNA Methylation: This process is crucial for regulating gene expression, where methylation patterns can influence whether genes are turned on or off. It plays a significant role in cellular differentiation by establishing and maintaining cell identity, which can indeed stabilize viral latency by silencing viral genes.
• Histone Modifications: These modifications, including methylation, acetylation, and others, alter chromatin structure, affecting gene accessibility and thus expression. They are integral to the epigenetic regulation of both host and viral genes, influencing cell fate decisions and potentially maintaining viral latency.
• Protein-Protein Interactions: These are fundamental to all cellular processes, including those that regulate the cell cycle, gene expression, and immune responses. They can directly impact how viruses interact with host cells, potentially enabling or maintaining viral latency.
• Light-Matter Interactions: While less directly connected in traditional molecular biology explanations, light can influence biological processes, including circadian rhythms that affect gene expression, which might indirectly relate to cellular differentiation and viral behavior.
• miRNA-mRNA Interactions: miRNAs are key regulators in post-transcriptional control, affecting mRNA stability and translation. Their roles in modulating host-pathogen interactions are well-documented, including in viral latency where miRNAs can target both host and viral mRNAs to regulate the viral lifecycle.

However, to say these mechanisms link to “every aspect” of cell type creation and differentiation across kingdoms via these interactions might be an oversimplification:

• Complexity Across Kingdoms: The mechanisms of cellular differentiation and the role of epigenetic control can vary significantly across different species and kingdoms. For instance, the specifics of how these mechanisms operate in bacteria, plants, and animals differ due to evolutionary divergence.
• Interplay and Redundancy: While these mechanisms are indeed interconnected and play roles in cellular differentiation, there’s considerable interplay and sometimes redundancy among them. Not all differentiation pathways in all cell types or species will rely equally on each mechanism.
• Viral Latency: The maintenance of viral latency is just one aspect of cellular regulation where these mechanisms play a role. There are many other factors, including immune responses, environmental conditions, and genetic predispositions, that also influence cell differentiation and type creation.

In summary, these mechanisms are essential but part of a broader, more complex network of biological regulation. The assertion that they cover “every aspect” through the mechanisms you describe would require comprehensive evidence across all life forms and conditions, which the current scientific literature does not fully support in such an absolute manner.

My response: The current scientific literature supports my claims about the mechanisms in 178,200 results from today’s search for miRNAs.

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