control of homeostasis by the epigenetic effects of proper nutrition and social stress inhibition might prevent our diet-driven and social stress-driven extinction.
I will not dismiss the potential importance of oxytocin to effective treatment despite the fact that the mechanisms of its effectiveness remain unknown.
What I don’t see is anyone who is integrating the ecological, social, and neurogenic niches and considering the epigenetic effects of nutrient chemicals and pheromones in the context of endocrine disruption and transgenerational epigenetic inheritance.
I may be confused about proximate and ultimate cause, especially if the role of transgenerational epigenetic inheritance is not central to the extended evolutionary synthesis. Isn’t transgenerational epigenetic inheritance the problem that led Dickins and Rahman to suggest a thought experiment; one where epigenetic mechanisms introduce shifts in learning bias for certain associations as would endocrine functioning? If so, my model (Kohl, 2012), which combines the epigenetic effects of nutrient chemicals and pheromones with biased endocrine functioning and adaptive evolution,…
This fact can be explained to a general audience by saying that food odors cause us to eat what causes us to produce pheromones that cause us to associate, or not associate, with other people. In this context, pheromones are social odors.