Our work demonstrates one can introduce an active control to DNA conductance by modifying a base with a redox group, and switch the DNA conductance reversibly between two levels by oxidizing or reducing the redox group with an EC gate. This strategy could be implemented in more sophisticated DNA nanostructures for active device building blocks. As the DNA conductance is an indicator of the molecule in the reduction or oxidation state, it is possible to study redox reaction kinetics at the single-molecule level by monitoring the DNA conductance.
…the engineered DNA provides a nice tool to examine redox reaction kinetics, and thermodynamics the single molecule level…
This means they can examine virus-driven energy theft in the context of redox reaction kinetics and thermodynamics at the single molecule level, which links nutrient energy-dependent changes from angstroms to ecosystems via the physiology of pheromone-controlled reproduction in species from microbes to humans. Simply put, they can link virus-driven energy theft from mutations to all pathology in species from archaea to modern humans.
The virus is made up of tiny biological computers called “noocytes,” where were intended to improve the human body — giving it routine maintenance and maximizing human potential. Instead, it wiped out most of North America.
“The US is the number one donor in the work that we do. Government aid can’t be replaced by philanthropy,” the 61-year-old told the Guardian.
Under the order, which is also known as the Mexico City Policy after it was first unveiled at a UN conference there in 1984, no government funding for family planning services can be given to clinics or groups outside the US that offer abortion or counselling services.
Why is Bill Gates condemning any of President Trump’s foreign policies before the Bill and Melinda Gates foundation stops funding research that is irrelevant to prevention of the viral apocalypse. Does Bill Gates decide who gets the vaccine and who doesn’t at the time the apocalypse begins, or will other billionaires like George Soros dictate all foreign policies? Who are you going to trust with the lives of your loved ones?
See also: Leakers beware: Trump has utterly defeated the disloyal coteries of the US intelligence community with my emphasis
The Trump administration exposed the false flag terrorism in the media.
Donald Trump posed as a hit man. He made sure that the leakers in the IC knew that their actions were illegal. He WARNED them. Then he launched his sting.
Donald Trump gave a press conference in which he admitted to the sting. He said that the leaks are real, but the news is fake.
It was his tactic, and he had to expose how he used it. That exemplifies Trump’s brilliance. Any intelligent disloyal Democrat from the intelligence community should have seen it coming. That suggests none of them are intelligent adversaries.
The biased media reporting on the press conference attests to lack of intelligence among the media representatives who reported the claims of Trump’s ignorant adversaries. The media representatives continue to look more foolish every time they speak out against the President of the United States.
The media representatives won’t be charged with any crimes. How many disloyal Democrats from the intelligence community will be prosecuted for treason?
How will President Trump deal with disloyal billionaires and others who waste his time attempting to influence the people of the United States and cause them to challenge him on topics they know nothing about?
Who will be the next billionaire to inadvertently refute theistic evolution by linking what is known about nutrient energy-dependent RNA-mediated amino acid substitutions to healthy longevity and linking virus-driven energy theft to all pathology?
Will Mark Zuckerberg eliminate the false flag terrorist groups from Facebook?
Will Paul Allen stop funding brain research that does not link the speed of light on contact with water to all energy-dependent cell type differentiation in all living genera via biophotonics and optogenetics?
Darwin introduced a viewpoint that was radically unsettling: we don’t progress to a more perfect form, but adapt to local environments. If humans are machines, then we can simply repair the broken parts. But if there is something more fundamental to the crisis of life than mere mechanisms of biology, then risk, and an element of danger, will always be with us.
Humans are not machines. Bill Gates probably knows that. And he probably knows that theistic evolution is the threat that can now be placed into the context of Non-theistic evolution
The major criticism of theistic evolution by non-theistic evolutionists focuses on its essential belief in a supernatural creator. These critics argue that by the application of Occam’s razor, sufficient explanation of the phenomena of evolution is provided by natural processes (in particular, natural selection), and the intervention or direction of a supernatural entity is not required. Evolutionary biologist Richard Dawkins considers theistic evolution a superfluous attempt to “smuggle God in by the back door”.
God doesn’t need to be smuggled in. He’s always been with those who believe. Believers know that natural selection for energy-dependent codon optimality occurs in the context of the physiology of pheromone-controlled reproduction. The physiology of energy-dependent reproduction links natural processes to fixation of amino acid substitutions in supercoiled DNA that differentiate the cell types of all living genera.
Supercoiled DNA protects all organized genomes from virus driven energy theft, which is linked to all pathology by mutations. Theistic evolutionists may continue to claim that they believe in mutation-driven evolution, but that’s because they known nothing about energy-dependent top-down causation.
Ask Bill Gates or a theistic evolutionist where the energy comes from, and see for comparison.
CARTs are structurally unique and operate through an unprecedented mechanism, serving initially as oligo(alpha-amino ester) cations that complex, protect, and deliver mRNA and then change physical properties through a degradative, charge-neutralizing intramolecular rearrangement, leading to intracellular release of functional mRNA and highly efficient protein translation.
Reported as: A new way Forward for Gene Therapy
Sickle-cell disease is caused by a mutation that links the transgenerational epigenetic inheritance of virus-driven energy theft to failed nutrient energy-dependent RNA-mediated DNA repair. Hemoglobin S is a naturally occurring hemoglobin variant — one of more than 1200 other variants. The variants link RNA-mediated amino acid substitutions in the cell types of populations of ecologically adapted humans. Clearly, their lineages adapted to ecological variation in the parts of the world where they were raised. Researchers have delivered nutrient energy-dependent messenger RNA (mRNA) into cells that use the mRNA to make proteins. What’s missing from this report on gene therapy are facts about how nutrient energy-dependent viral latency must be linked to healthy longevity via energy-dependent changes in the microRNA/messenger RNA balance linked to amino acid substitutions in supercoiled DNA, or from virus-driven energy theft to all pathology.
I continue to encourage comments from those who are not Combating Evolution to Fight Disease, especially if they are willing to tell others what they like about virus-driven energy theft and all pathology. Now that Bill Gates has clearly stated that virus-driven energy theft is not likely to be a good thing for humanity, perhaps he will join the serious scientists and/or help to fund their works.
See for example: The 2000 T. H. Morgan Medal Essay: H. J. Muller and the Nature of the Gene and works published by Ruth Ann Luna.
For example, The Brain-Gut-Microbiome Axis: What Role Does It Play in Autism Spectrum Disorder? and her presentation from February 22, 2017:
She graciously answered these three questions:
Q: Is the gut microbiome the most likely link from nutrient energy-dependent metabolic networks to genetic networks and Precision Medicine via the National Microbiome Initiative?
Ruth Ann Luna PhD, MB (ASCP) CM
It’s certainly a key player in the crosstalk, but there’s still much to uncover about these pathways.
Q: Have you placed the 2016 publication of “Codon identity regulates mRNA stability and translation efficiency during the maternal-to-zygotic transition” into the context of natural selection for energy-dependent codon optimality and transgenerational epigenetic inheritance in your works?
Ruth Ann Luna PhD, MB (ASCP) CM
No we have not. We are working from functional gut microbiome up the gut-brain axis at this point and hoping to weave in as many other factors as possible.
Q: Who else is addressing the bidirectional communication besides your group?
Ruth Ann Luna PhD, MB (ASCP) CM
There are many other groups evaluating the gut-brain connection in general, but not necessarily exclusive to ASD. I’d suggest a quick search in Pubmed for the latest groups involved in this area.
Her expressed intent to “weave in other factors that already are known to me and to Teresa Binstock inspired me to perform this search for the terms autism and microRNA
See for example from February 17, 2017: Regulation of mRNA splicing by MeCP2 via epigenetic modifications in the brain
Our analysis thus indicated that MeCP2-mediated alternative splicing might influence neuronal functions via two different strategies: one is to regulate protein diversity by coding exons, and another is to regulate protein expression by non-coding exons. Our studies not only systematically explore the mechanisms underlying MeCP2-mediated alternative splicing, but also provide insights into the roles of MeCP2-mediated alternative splicing, which could influence both protein diversity and protein expression level in neurons.
Our 1996 Hormones and Behavior review explored the molecular mechanisms of RNA-mediated alternative splicings and nutrient-dependent pheromone-controlled biophysically constrained protein folding chemistry in the context of the physiology of reproduction in species from microbes to humans.
See our molecular epigenetics section in From Fertilization to Adult Sexual Behavior
Small intranuclear proteins also participate in generating alternative splicing techniques of pre-mRNA and, by this mechanism, contribute to sexual differentiation in at least two species, Drosophila melanogaster and Caenorhabditis elegans (Adler and Hajduk, 1994; de Bono, Zarkower, and Hodgkin, 1995; Ge, Zuo, and Manley, 1991; Green, 1991; Parkhurst and Meneely, 1994; Wilkins, 1995; Wolfner, 1988). That similar proteins perform functions in humans suggests the possibility that some human sex differences may arise from alternative splicings of otherwise identical genes.
If my co-authors and I had been funded to link what was known about biologically-based cell type differentiation in all species to their origins and from ecological variation to ecological adaptations in all living genera, you would not need to ask whether or not some or all of your loved ones will survive the viral apocalypse.
Nutrient energy-dependent sexual differentiation in yeasts at the advent of sexual reproduction has been linked to all cell type differentiation in all individuals of all species via the conserved molecular mechanisms of pheromone-controlled reproduction that we began to detail two decades ago.
Our findings of sexual dimorphism in sncRNA levels underscore the importance of considering sex, in addition to age, when interpreting molecular findings on ASD brain.
The interpretation of molecular findings on ASD brain can be placed into the context of virus-driven energy theft and all pathology via the transgenerational epigenetic inheritance of Zika virus-damaged DNA in humans. For comparison see how Greg Bear used information on nutrient-dependent pheromone-controlled RNA-mediated protein folding chemistry to ecological adaptations in a new human subspecies.
…etiological models suggest that the biological male phenotype carries a higher intrinsic risk for ASD than the female phenotype. To our knowledge, this hypothesis has never been tested directly, and the neurobiological mechanisms that modulate ASD risk in male individuals and female individuals remain elusive.
The neurobiological mechanisms link sex differences in the cell types of yeasts to sex differences in virus-driven energy theft and all pathology.
Reviewed as: Evolution rising from the grave
Bear goes a little further in suggesting that such change can occur over about a generation, an idea that might be a little too radical at the moment. However, he does mention data suggesting that fruitflies can adapt to a new environment in just a few generations of selection.
Reviewed as: Living with the Neanderthals
Bear’s two Darwin novels were not written just to entertain. He also seeks to teach readers about science, to highlight our utilitarian politics and our inability to get along with each other, and to provide a quasi-rational basis for theology and morality. He advances a world view in which we are all part of the vast neural network of life, cutting across ethnic borders, species divides and the chasms between taxonomic kingdoms, in balance, and in two-way communication, with the ecosystem.
Bear qualifies to win the next Nobel Peace Prize, or minimally, the Prize for Literature. When you realize whose information is being used as a basis for the claims by Bill Gates that support young earth creationism, see also: Viral Genome Junk Is Bunk
…in an ironic twist, the evidence mentioned above indicates that viruses likely arose from their hosts and not the other way around. As molecular biologist and biochemist Peter Borger notes, “The most parsimonious answer is: the RNA viruses got their genes from their hosts. 6
Where else would viruses get their genes, and what else would force organisms to use the sun’s anti-entropic virucidal energy to adapt?