From Michigan State University

Ask the expert: What is Juneteenth?

Historical Perspective:
See: “Apology to People of Color for APA’s Role in Promoting, Perpetuating, and Failing to Challenge Racism, Racial Discrimination, and Human Hierarchy in U.S.” October 29, 2021

When will this historic apology be celebrated? Did you even know about it?

Michigan State’s Richard Lenski still promotes the moronic theory that racial differences evolved from nutrient-dependent pheromone-regulated genetic processes of reproduction in E. coli.

See: Experimental Test of the Contributions of Initial Variation and New Mutations to Adaptive Evolution in a Novel Environment 6/1/22

For comparison, light-activated biophotons in all cell types at the pH-dependent origin of life ~6000 years ago links peptide synthesis to “Footprints of a Singular 22-Nucleotide RNA Ring at the Origin of Life” 4/25/20 via

“… a model revisiting the architecture of RNA-peptide interactions…”

Juneteenth is merely an attempt made by social scientists, pseudoscientists, and other gene-centric theorists  to placate the ignorant masses by making it appear that Democrats, Communists, and other atheists want to end racism. The Democratic party thrives on that level of ignorance.

Historical perspective

In 1999, I presented during the Human Behavior and Evolution Society’s 11th Annual Conference, See pages 18, 44 and 61 of the program

Human pheromones and physical attraction: More than meets the eye.

Visual aspects of human physical attraction, (e.g., hair color; amount and location of terminal hair; breast development; assessment of age, height, and weight) have no readily apparent counterpart in other mammals. These aspects of physical attraction correlate with genetic and/or steroidogenic variability. Genetic and steroidogenic variability correlate well with mammalian pheromone production and distribution. Mammalian pheromones act on a prenatally predisposed neuroendocrine sequence that has been linked to behavior. This sexually dimorphic neuroendocrine sequence allows pheromones to elicit functional changes in pituitary and gonadal hormone secretion. During development, these changes direct the concurrent maturation of the neuroendocrine system, the central nervous system, and the reproductive system. These systems coordinate properly timed reproductive sexual behavior.
Human pheromones alter pituitary and gonadal hormone secretion. Comparing the effects of visual and olfactory input on hormonal correlates of physical attraction provides evidence that there is more to human physical attraction than meets the eye. This integrative literature review suggests that a pheromonally driven stimulus-response cycle, which is common to many species, conditions human physical attraction to visual stimuli.

In 1999, I also presented “Mammalian Olfactory-Genetic-Neuronal-Hormonal-Behavioral Reciprocity and Human Sexuality

The early prenatal migration of gonadotropin releasing hormone (GnRH) neurosecretory neurons into the brain enables mammalian olfactory pathways to exhibit sexually dimorphic specificity to social-environmental chemical stimuli (e.g., pheromones). Mammalian pheromones induce change in GnRH pulsatility. Hypothalamic GnRH pulsatility directs the concurrent maturation of the neuroendocrine, central nervous, and reproductive systems. Pheromonally induced alterations in GnRH pulsatility allow for a life-long causal linkage between pheromones, olfaction, gene activation, neurotransmission, autonomic responses, luteinizing hormone/follicle stimulating hormone ratios, steroidogenesis, synaptogenesis, synaptolysis, apoptosis, and hormonally induced behavioral changes. Human pheromones induce changes in pituitary and gonadal hormone secretion, apparently by influencing GnRH pulsatility. Accordingly, human pheromones appear to link the genetic “nature” and the social-environmental “nurture” of human reproductive sexual behaviors and of other behaviors via a conditioned stimulus-response cycle common to many species.

“Olfactory/pheromonal input and human female proceptive sexual behaviors/preferences” was presented at the 2008 Annual meeting of the Society for Neuroscience in Washington, DC.

Human pheromones increase women’s flirtation behavior during the 13th Annual Meeting of the Society for Behavioral Neuroendocrinology at Michigan State University in June 2009, and again at the Atlanta Chapter of the Society for Neuroscience at Emory University on 10/13/09, which is very close to the headquarters of the Center for Disease Control (CDC) in Atlanta where Timothy J. Cunningham suddenly disappeared in 2018 (He was subsequently found drowned).

See also this proof of concept presented during the International Society for Human Ethology’s 2010 meeting in Madison, Wisconsin: Human pheromones increase women’s observed flirtatious behaviors and ratings of attraction

Social-environmental odor cues, which conspecifics learn to associate with sexual activity, induce changes in the hypothalamic-pituitary-gonadal axis. Classical conditioning may evoke functional changes in the neuroendocrine pathways that mediate the release of luteinizing hormone (LH), and associated changes in behavior. Mammalian conditioning paradigms suggest that androsterone is a human chemical signal of reproductive fitness, which could be unconsciously associated: (1) with an LH response to androstenol; (2) with other sensory input from the social environment, and (3) with behavior. During a cooperative task, we controlled for variability in non-olfactory/pheromonal sensory input from the social environment, and varied the presence or absence of the chemical mixture that our accomplice applied. We observed statistically significant increases in women’s flirtatious behaviors when our accomplice was wearing the androsterone/androstenol mixture, and women’s self-reported level of attraction to our accomplice increased. The neurophysiological effect and the associated behavioral affect meet criteria that characterize human pheromones.

“Human pheromones and nutrient chemicals: epigenetic effects on ecological, social, and neurogenic niches that affect behavior” was presented at the 2012 Society for Social Neuroscience annual meeting in New Orleans, LA.

See also this 2010 slide presentation It was subsequently linked from Modelling biological behaviours with the unified modelling language: an immunological case study and critique 10/6/14 to Stress-induced mutagenesis and complex adaptation 10/7/14 and to “The aroma of arousal: Effects of menstrual cycle phase and women’s sexual arousal state on men’s responsiveness to women’s body odor” 3/1/19 via the nutrient-dependent pheromone-regulated genetic processes of reproduction in species from microbes to humans and Thomas E. Ichim’s patented therapeutic solutions for the use of stem cells to repair virus-damaged DNA.

Compare the facts about photochemical Induction of mouse totipotent stem cells by a defined chemical cocktail 6/21/22 to any claim ever made by ignorant and arrogant neo-Darwinian theorists who refuse to start with light-activated carbon fixation in cyanobacteria and microRNA-mediated protein folding chemistry for comparison to this claim about sunlight and humidity made by former President Trump on 4/23/20.

See: USA today 4/23/20 Medical experts rip Trump’s suggestion that sunlight, disinfectants may treat coronavirus and the claim by Jonathan Reiner, professor of medicine at The George Washington University Hospital who said “there’s no medical basis for Trump’s statements about sunlight.”

Reiner’s claim has been removed from view on all, or nearly all, mass media outlets, but I have screenshots for anyone who needs proof of human idiocy.

See for comparison:

See also from my Society for Behavioral Neuroendocrinology conference in Atlanta, Georgia 6/24/13:

No photo description available.

Nothing about constraints on viruses has changed. But millions have suffered unnecessarily and died prematurely. WHO is responsible for that. Why did Biden put us back into their domain?

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