See: The Bull Sperm MicroRNAome and the Effect of Fescue Toxicosis on Sperm MicroRNA Expression

The links from the anti-entropic virucidal energy of the sun are obvious. Plant microRNAs link the physiology of pheromone-controlled reproduction in soil bacteria from the physiology of food energy-dependent reproduction in animals to the pheromone-controlled physiology of reproduction in all animals via biophysically constrained viral latency, which is mediated by the fixation of amino acid substitutions.

See Schrodinger (1944) What is life? In the forward to the reprint edition, Roger Penrose puts the facts succinctly.

How often do we still hear that quantum effects can have little relevance in the study of biology, or even that we eat food in order to gain energy?

See also: From Fertilization to Adult Sexual Behavior (1996)

Small intranuclear proteins also participate in generating alternative splicing techniques of pre-mRNA and, by this mechanism, contribute to sexual differentiation in at least two species, Drosophila melanogaster and Caenorhabditis elegans (Adler and Hajduk, 1994; de Bono, Zarkower, and Hodgkin, 1995; Ge, Zuo, and Manley, 1991; Green, 1991; Parkhurst and Meneely, 1994; Wilkins, 1995; Wolfner, 1988). That similar proteins perform functions in humans suggests the possibility that some human sex differences may arise from alternative splicings of otherwise identical genes.

See for comparison: The Expanding Landscape of Alternative Splicing Variation in Human Populations (2018)

The prevalent role of alternative splicing in Mendelian and complex diseases suggests that evaluating the impact of genomic variants on splicing needs to be an integral part of clinical variant prioritization. Many computational tools and online resources exist for prioritizing and annotating variants discovered by exome or genome sequencing.96 Most tools are designed to predict the pathogenic effects of missense variants on protein products. However, there is overwhelming evidence that missense, nonsense, and silent variants within exons, as well as intronic variants, can disrupt splicing and cause disease.14

After accurate representations of how food energy-dependent alternative splicing variation in all species has been linked to biophysically constrained viral latency during the 22 years since book publication of The Scent of Eros: Mysteries of Odor in Human Sexuality, this pseudoscientific nonsense about evolution was published.

The human microbiome in evolution

It is often implied that humans and their microbes have adapted to each other, and that perturbing this relationship results in disease. Evidence suggesting non-neutral processes exists in a few cases [54, 58]. We must demonstrate fitness consequences in these cases when adaptation is assumed—and be open to non-adaptive explanations for health-relevant phenotypes (Box 3).

Population genetics theory plays a pivotal role in defining expectations in evolutionary studies, including host–pathogen interactions [104, 105, 106, 107]. Additionally, the community genetics framework assesses the effect of a particular organism’s heritable traits on the ecosystem more broadly [108]. There is strong motivation to model evolutionary processes of host–microbiome systems perhaps borrowing from some of these approaches.

No serious scientist has ever assumed that ecological adaptation automagically occurs. No serious scientist, since Dobzhansky (1973), has implied that ecological variation can be linked to ecological adaptation by evolution across millions of years.

Population genetics theory bastardized Darwin’s claims about conditions of food energy-dependent biophysically constrained life, and the population genetics have caused all unnecessary suffering and premature death. The population geneticists are not motivated to stop killing people by admitting that their theories have always been ridiculed by serious scientists because some of the serious scientists are creationists.

Evolutionary theorists and “Big Bang” cosmologists do not want to let a creationist foot in the door.

…we are forced by our a priori adherence to material causes to create an apparatus of investigation and a set of concepts that produce material explanations, no matter how counter-intuitive, no matter how mystifying to the uninitiated. Moreover, that materialism is absolute, for we cannot allow a Divine Foot in the door. — Richard Lewontin

Serious scientists have collaborated to kick down that door by helping to develop the cell biology game “Cytosis.”

A board game taking place inside a human cell! Players compete to build enzymes, hormones and receptors and fend off attacking Viruses!

The game can be used to teach players who are 10+ years of age how to link biophysically constrained viral latency to all biodiversity by starting with the creation of ATP. The question expected to immediately arise is: Where did the energy to create ATP come from.

One answer has already been placed into the context of the evolution of differences in hydrogen.

Chemists reveal one mechanism of dihydrogen production by nitrogenase

This work is a critical step toward a mechanistic understanding of the nitrogenase enzyme. There is still work to be done to understand the mechanism of the other H2 evolution process that leads to N2 binding, activation, and reduction to NH3.

If you believe that differences in hydrogen atoms evolved, you may want to continue to believe that the differences in the energy of the hydrogen atoms emerged. But no serious scientists will listen to your pseudoscientific nonsense about emergence and evolution.

But anyone age 10+ who has played the cell biology game “Cytosis” will laugh at you. If you do not want your children and grandchildren to be bullied by students who are taught to believe in experimental evidence of biophysically constrained viral latency, advise them to say nothing about mutations, natural selection, or evolution in the schoolyard.

In the context of what is known about enzyme-constrained interethnic biodiversity, watch what happens to the value of these stocks, which is largely based on claims about the benefits of gene editing.

On January 8, 2018, you could have seen the first indication that investors may not be willing to continue being bamboozled.

Symbol Price Change Change %
NTLA 20.03 -2.68 -11.80%
CRSP 26.07 -0.74 -2.76%
EDIT 30.02 -3.57 -10.63%


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