Several previous studies have shown that host miRNAs play an anti-viral role and improve the treatment of patients with COVID-19. miRNAs by binding to the 3′-UTR or 5′-UTR of viral RNA play an important role in COVID-19-host interplay and viral replication.
That fact makes it easier to convict Facebook of crimes against humanity because their censors prevent me from including links to autophagy.pro, and to microRNA.pro which expose the scientific truths about viral replication on RNA-mediated.com.
Understanding how energy-dependent microRNA-mediated autophagy is linked to biophysically constrained viral latency via RNA interference (RNAi) is essential to disease prevention and/or effective treatment.
…miR-340-5p can be exploited for removing senescent cells to restore tissue homeostasis and mitigate damage by senescent cells in pathologies of human aging.
You need only learn about God’s energy-dependent biophysically constrained Creation of the lamin B receptor and other proteins. One change in a base pair makes the difference between mutations and adaptations.
rs11614913 is a polymorphism affecting function of miR-196a in complex diseases.
This base pair change can be linked to chromosomal rearrangements, which protect organized genomes from viruses in species from microbes to humans. This includes innate protection from the SARS-CoV-2 spike antigen and COVID-19.
See for comparison: “One crank dies, another rises to take his place“(2014)
Behold James Vaughn Kohl.
Ecological adaptation occurs via the epigenetic effects of nutrients on alternative splicings of pre-mRNA which result in amino acid substitutions that differentiate all cell types of all individuals of all species. The control of the differences in cell types occurs via the metabolism of the nutrients to chemical signals that control the physiology of reproduction.
That accurate representation of top-down causation has been linked to: Immunodominant T-cell epitopes from the SARS-CoV-2 spike antigen reveal robust pre-existing T-cell immunity in unexposed individuals 6/23/21
…findings suggest that SARS-CoV-2 reactive T-cells are likely to be present in many individuals because of prior exposure to flu and CMV viruses.
Pre-existing immunity suggests that SARSCoV2 was genetically engineered for population control via death of the debilitated. Intelligent serious scientists know that death could be caused by one virus-driven change in a single amino acid substitution in the SARS-CoV-2 spike antigen. They have known that since 11/22/13. For instance: Koel et al. (p. 976) show that major antigenic change can be caused by single amino acid substitutions.
For comparison, biophysically constrained pre-existing immunity was linked to the end of the 1918 influenza: See: A two-amino acid change in the hemagglutinin of the 1918 influenza virus abolishes transmission
Behold Nate Weymouth.
In this discussion attempt, I showed that he is a biologically uninformed science idiot, like PZ Myers.
JK: All evidence of biophysically constrained viral latency across kingdoms supports the claim that the variant was purposefully genetically engineered. Biophysically constrained human endogenous retroviruses typically protect us, unless we are convinced to inject synthetic mRNA.
NW: Containing viral latency is no evidence Cov2 was genetically engineered. Familiar with GA research community, not aware of your papers, curious. If concerned about mRNA vaccines, measure Cov2 antibody levels, might have immunity already. Over interpretation of the paper.
My papers and presentations link “Visualizing a protonated RNA state that modulates microRNA-21 maturation” to Peptide synthesis at the origin of life via biophysically constrained human endogenous retroviruses (HERVs). You fool!
NW: I worked at UNC Chapel Hill. Don’t see your name on these papers? If you lived in GA & work as a virologist, only a few places you can work. I will read the papers but think you’re taking credit for something you didn’t do & misinterpreting the findings of the quoted papers.
Nate Weymouth published GFP-Moesin Illuminates Actin Cytoskeleton Dynamics in Living Tissue and Demonstrates Cell Shape Changes during Morphogenesis in Drosophila 11/1/97 after my group linked God’s energy-dependent Creation of stem cells from microRNA biogenesis to RNA interference and biophysically constrained viral latency across kingdoms in From Fertilization to Adult Sexual Behavior (1996).
See: The polyribosomal protein bound to the 3′ end of histone mRNA can function in histone pre-mRNA processing (1995) I co-authored a book about this in 1995.
See RNA-Guided Human Genome Engineering You could have exposed that fact. Instead I’ve exposed you.
See: microRNA-mediated replication timing (4)