…deadly coral diseases were 50% less prevalent in regions with seagrass meadows. The scientists aren’t entirely sure how the seagrass combats bacteria so effectively, but one possibility could be that it anchors the nutrient-rich sediment to the sea floor, effectively preventing harmful microbes from feeding on the nutrients.
They seem to be claiming that scientists do not know how to link virus-driven energy theft from the viral hecatomb to all pathology in species from marine bacteria to humans. How can serious scientists not be sure about this?
We show here for the first time the crucial role of viruses in controlling archaeal dynamics and therefore the functioning of deep-sea ecosystems, and suggest that virus-archaea interactions play a central role in global biogeochemical cycles.
That suggestion can be linked to the claim that bacteria living in ocean sediments had reportedly not changed in ~2 billion years. If the speed of light on contact with water limits the virus-archaea interactions, the anti-entropic virucidal effect of sunlight probably plays the most important role in global biogeochemical cycles. The stability of the global biogeochemical cycles can be placed into the context of claims about evolution.
The greatest absence of evolution ever reported has been discovered by an international group of scientists: a type of deep-sea microorganism that appears not to have evolved over more than 2 billion years. But the researchers say that the organisms’ lack of evolution actually supports Charles Darwin’s theory of evolution.
Darwin clearly stated that “conditions of life” must be considered before natural selection. These researchers reported that the “conditions of life” did not change, which meant their was not reason for nutrient-dependent pheromone-controlled ecological adaptation to occur in the context of the physiology of reproduction in bacteria living in ocean sediments. The claims that they bacteria had lived there for more than 2 billion years have never been supported by experimental evidence. Instead, experimental evidence has repeatedly refuted those ridiculous claims,
From the Editor’s Summary
Losing and then regaining flagella
The ability to adapt to changes in the function of gene regulators, as opposed to structural genes, is a crucial aspect of evolutionary change. Taylor et al. mutated a central regulator for the formation of flagella in the bacterium Pseudomonas fluorescens. They then put the mutated flagella-free bacteria under strong selection pressure to regain mobility. The mutated bacteria regained the lost flagella, and motility, within 4 days.
The lost flagella could not have been regained outside the context of the nutrient energy-dependent biophysically constrained pheromone-controlled physiology of reproduction that links supercoiled DNA to protection of all organized genomes from virus-driven energy theft and genomic entropy. The supercoiled DNA also links the Virus-mediated archaeal hecatomb in the deep seafloor to the energy-dependent pheromone-controlled stability of the global biogeochemical cycles in the context of the physiology of reproduction via quorum sensing in bacteria and the energy-dependent pheromone-controlled physiology of reproduction in species from microbes to humans.
See also: Guide RNA
The facts about RNA editing link nutrient energy-dependent microRNA flanking sequences from hydrogen-atom transfer in DNA base pairs in solution to all biophysically constrained biodiversity via the physiology of reproduction in all living genera. For more evidence that virus-driven energy theft is linked to all pathology, see:
We also showed that miR-15a-5p could bind to the CDKN2B 3’UTR, resulting in a significant decrease in luciferase activity compared with the scramble control. Furthermore, we found that the cells isolated from AAA participants showed an over-expression of miR-15a-5p compared to the normal controls, while the CDKN2B mRNA and protein expression level of the AAA group were much lower than the normal control group. Additionally, the expression of CDKN2B mRNA and the protein of the cells transfected with miR-15a-5p mimics and CDKN2B siRNA was downregulated, while the cells showed upregulated expression subsequent to transfection with miR-15a-5p inhibitors compared to the scramble control.