People who understand the facts about how energy-dependent ecological adaptations to viruses across kingdoms and across generations prevent pathology do not need more theoretical models.
For instance, see: Genome-Wide Study Reveals a Novel Regulatory Pathway: Translation Affects mRNA Stability in a Codon-Dependent Manner on June 26, 2019.
It is time to force theorists to move forward. Trump-hating advocates of vaccines + GMOs (2)
For an attempt to return to the neo-Darwinian pseudoscientific nonsense of the past, see: Neuronal Small RNAs Control Behavior Transgenerationally (6/6/19)
…we propose a small-RNA-based mechanism for communication of neuronal processes transgenerationally.
They validated my model of how light-activated energy-dependent microRNA-mediated transgenerational inheritance must be linked to all morphological and behavioral phenotypes via sympatric speciaton in nematodes.
See for comparison: Principles of Transgenerational Small RNA Inheritance in Caenorhabditis elegans (7/24/17)
The Principles of Transgenrational Small RNA Inheritance were included in our section on molecular epigenetics from a 1996 Hormones and Behavior review: From Fertilization to Adult Sexual Behavior
Small intranuclear proteins also participate in generating alternative splicing techniques of pre-mRNA and, by this mechanism, contribute to sexual differentiation in at least two species, Drosophila melanogaster and Caenorhabditis elegans (Adler and Hajduk, 1994; de Bono, Zarkower, and Hodgkin, 1995; Ge, Zuo, and Manley, 1991; Green, 1991; Parkhurst and Meneely, 1994; Wilkins, 1995; Wolfner, 1988). That similar proteins perform functions in humans suggests the possibility that some human sex differences may arise from alternative splicings of otherwise identical genes.
On 7/27/18 Oded Rechavi blocked me on Twitter after he reported: A Molecular Grammar Governing the Driving Forces for Phase Separation of Prion-like RNA Binding Proteins on 7/26/18
My response: Thanks. Most people missed the claim that achiral glycine in position 6 of the GnRH decapeptide was the link to the food energy-dependent pheromone-controlled stability of the organized mammalian genome. That helps to explain their lack of scientific progress compared to your[s].
When he realized I had linked alternative splicings of pre-mRNAs, which are now called microRNAs, from one amino acid substitution, namely EDAR V370A to the food energy-dependent pheromone-controlled stability of the organized mammalian genome, and biophysically constrained viral latency in humans via the mouse model organism, Oded Rechavi blocked me from seeing his tweets.
That made it obvious to me that he had actually missed the claims about achiral glycine, and I suspected that he was going to plagiarize my life’s work and pretend my model of biophysically constrained viral latency fit into the context of neo-Darwinian evolution.
Now, my life’s work shows up in his latest work, which was reported as: Study shows how the nervous system can transmit information across multiple generations
…parents could potentially transmit information that would be beneficial to the progeny in the context of natural selection. It could therefore potentially influence an organism’s evolutionary course.
Natural selection for energy-dependent codon optimality cannot be linked from alternative splicings of pre-mRNAs/microRNAs via any organism’s evolutionary course. Ecological adaptations require the link from sunlight to the creation of ATP and the link from the creation of ATP to the creation of RNA and RNA interference, which biophysically constrains viral latency. Without the constraints, the virus-driven degradation of messenger RNA links mutations to all pathology.
Obviously, his entire group failed their “Ecology and Adaptation” courses. Rechavi and his group, want to continue to tout neo-Darwinian evolution.
As a researcher who plagiarized my model, he also appears to be a traitor to his country, or minimally the Israeli school system.
…learning about evolution is not the primary function of the decision, but rather to use it as a building block for students to learn more about their ecology.
Other researchers from Israel reported ecological adaptations across kingdoms in the context of measurements of fluorescence linked to the cure for all cancer.
The cure links natural selection for energy-dependent codon optimality in P. fluorescens to biophysically constrained viral latency, which contributes to plant growth via sympatric speciation in soil bacteria.
I show illuminating examples of swarming intelligence of live bacteria in which they solve optimization problems that are beyond what human beings can solve. This will lead to a discussion about the special nature of bacterial computational principles compared to Turing algorithm computational principles, in particular about the role of distributed information processing.
Other researchers from Israel have led the way forward with scientific experiments that link the light-activated assembly of the microRNA-RNA-peptide nanocomplex from natural selection for energy-dependent codon optimality via RNA interference, which has consistently been linked from viral latency to healthy longevity and all biodiversity via the physiology of reproduction and transgenerational epigenetic inheritance.
Oded Rechavi’s group reversed the course with their claim about natural selection. I reiterate:
It could therefore potentially influence an organism’s evolutionary course.
Darwin placed his “conditions of life”first. All serious scientists have placed natural selection second because organisms that cannot find enough food do not reproduce. Natural selection must put selection for food before any evolutionary course. When researchers fail to do that, they exemplify human idiocy.
See for comparison: Age-dependent changes and biomarkers of aging in Caenorhabditis elegans (2/9/19)
Scientific creationists from South Korea have also linked the creation of the sun’s anti-entropic virucidal energy from microRNA biogenesis in plants to biophysically constrained viral latency and healthy longevity in all living genera.
“…we report for the first time that intercellular viral oncomiR spreading is an additional mechanism employed by KSHV to inhibit host anti-viral immunity and consequently promote oncogenesis.”
Rechavi is co-author of this article on virus-induced pathology, and still the author’s missed the link to the microRNA-mediated cancer cure via the measurement of fluorescence.
Missing the links is what neo-Darwinian theorists do. They haven’t found any missing links because serious scientists link the creation of sunlight to ecological adaptations via RNA interference and biophysically constrained viral latency.
See also: Turkish schools to stop teaching evolution, official says (2017)
“There are those for whom evolution is a religion and are unwilling to hear about anything else. Part of my responsibility, in light of my position with the Education Ministry, is to examine textbooks and curricula.”
See for comparison: GMOs are killing us: Facts you probably don’t know (6/5/19) and Trump Administration Proposes Easing Rules on Genetically Engineered Crops (6/5/19)
The overhaul, which the department said would cut the cost of developing genetically engineered plants, would exempt crops with traits “similar in kind” to modifications that could be produced through traditional breeding techniques. Developers would be allowed to make a “self-determination” that their products are exempt from regulation.
Any developer who uses viruses to alter the biophysically constrained energy of organisms from microbes to humans could be found guilty of crimes against nature in the context of natural selection for energy-dependent codon optimality, which prevents the virus-driven degradation of messenger RNA and the unrepaired DNA damage linked to mutations and pathology.