Estranged ‘White Coat Notes’ blogger exposes human idiocy (2)

By: James V. Kohl | Published on: June 18, 2018


…there are constraints on the mixed state entanglement…

The biophysical constraints attest to the fact that quantum entanglement in photosynthetic complexes does not evolve.

The works of two people who recently became members of the RNA-mediated FaceBook group attest to the overwhelming ignorance of biologically uninformed theorists.
See for example: The C. elegans hunchback Homolog, hbl-1, Controls Temporal Patterning and Is a Probable MicroRNA Target (2003) Chiara Gamberi (member of RNA-mediated) and Frank J. Slack are co-authors.
See also:  RNA-mediated gene activation (2013) Frank J. Slack is one of the two co-authors.
See also: rRNA—the evolution of that magic molecule (2015) Robin R. Gutell (member of RNA-mediated)

I learned that Carl (and my PhD adviser, Dr Harry Noller!) had the audacity to proclaim, “NEITHER, RNA came first.” Then later that fall I heard more diatribes. And this was published on the front cover of the reputable science journal, the New York Times, most likely to circumvent peer review.

Use of the New York Times to circumvent peer review led to the claims of Carl Zimmer in She Has Her Mother’s Laugh: The Powers, Perversions, and Potential of Heredity

“We need a new definition of what heredity is…”

See for comparison: The Surprising Origins of Life’s Complexity 7/16/13 and Nutrient-dependent/pheromone-controlled adaptive evolution: a model 6/14/13
Biologically uninformed so-called science journalists need only follow the extant literature and continue to ignore the facts. That facilitates the pseudoscientific nonsense they tout in their books, which are as meaningless as “Mutation-driven evolution.” 6/14/13
Theories exemplify human idiocy and most so-called science journalists believe in the ridiculous theories, or they simply earn a living by touting them.
What do you think that Carl Zimmer believes? See: How One Child’s Sickle Cell Mutation Helped Protect the World From Malaria
See for comparison: Single-trait and multi-trait genome-wide association analyses identify novel loci for blood pressure in African-ancestry populations
and Environmental selection during the last ice age on the mother-to-infant transmission of vitamin D and fatty acids through breast milk 4/23/18

The frequency of the human-specific EDAR V370A allele appears to be uniquely elevated in North and East Asian and New World populations…

The creation of the food energy-dependent human-specific allele links the mouse-to-human model of biophysically constrained pheromone-controlled viral latency to all biodiversity in species from microbes to humans.
See: Nutrient-dependent Pheromone-Controlled Ecological Adaptations: From Angstroms to Ecosystems
Concluding paragraphs:

The plausibility and ecological validity of Kohl’s Laws in the context of Darwin’s ‘conditions of life’ can be compared to theories about biologically-based cause and effect in the context of species diversity. In mammals, for example, the explanatory power of a model of ecological variation and biophysically constrained nutrient-dependent pheromone-controlled ecological adaptations became clear with companion papers published in 2013. See for review [26].

The companion papers [159,160] told a new short story of ecological adaptations. In the context of climate change and changes in diet, the story began with what probably was a nutrient-dependent base pair change and a variant epiallele that arose in a human population in what is now central China. Apparently, the effect of the epiallele was adaptive and it was manifested in the context of an effect on sweat, skin, hair, and teeth. In another mammal, such as the mouse, the effect on sweat, skin, hair, and teeth is probably due to a nutrient-dependent epigenetic effect on hormones responsible for the tweaking of immense gene networks that metabolize nutrients to pheromones.

The pheromones appear to control the nutrient-dependent epigenetically-effected hormone-dependent organization and hormone-activation of reproductive sexual behavior in mammals such as mice and humans, but also in invertebrates and in microbes as previously indicated. The ecological adaptations, which appear to be manifested in the human population are detailed in these two reports [159,160]. The ecological adaptations are likely to be nutrient-dependent and pheromone-controlled. If so, ecological variation probably leads to ecological, social, neurogenic, and socio-cognitive niche construction, which is manifested in increasing organismal complexity and species diversity. If not, there may be something as yet unknown about mutations and evolution that makes sense in the light of what is known about nutritional epigenetics and the molecular biology of species from microbes to man.

159. Kamberov YG, Wang S, Tan J, Gerbault P, Wark A, et al. (2013) Modeling recent human evolution in mice by expression of a selected EDAR variant. Cell 152: 691-702.
The selected EDAR variant is V370A
160. Grossman SR, Andersen KG, Shlyakhter I, Tabrizi S, Winnicki S, et al. (2013) Identifying recent adaptations in large-scale genomic data. Cell 152: 703-713.
V370A was placed into the context of how ecological variation must be linked to ecological adaptations in all living genera.
See also: C. Gamberi
For example: Human Gut Microbiota: Toward an Ecology of Disease.
See also: RR Gutell
For example: Structural constraints identified with covariation analysis in ribosomal RNA.
See for comparison: Sam Rodriques invents new technologies to help neuroscientists understand how the brain works
and Multipartite quantum entanglement evolution in photosynthetic complexes

Our results support the hypothesis that entanglement is maximum primary along the two distinct electronic energy transfer pathways. In addition, we note that the structure of multipartite entanglement is quite simple, suggesting that there are constraints on the mixed state entanglement…

The biophysical constraints attest to the fact that quantum entanglement in photosynthetic complexes does not evolve.

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