“New” quantum biology. Pirating the old
Jim Al-Khalili is a nuclear physicist. Luca Turin is a biologist.
Both presenters link Schrödinger’s claims in What is Life? Which one knows most about it?
Jim Al-Khalili frames his presentation in the context of evolutionary theory. See the text or the 20-minute Video presentation: Jim Al-Khalili: How quantum biology might explain life’s biggest questions
Luca Turin frames his presentation in the context of what is known to serious scientists about the biophysically constrained chemistry of nutrient-dependent RNA-mediated protein folding.
See also: Search Results for ‘”quantum biology”‘ and this history of how, since 1996, Luca Turin has continued to place his theory into the context of an atoms to ecosystems model of everything currently known about cell type differentiation in all cell types of all individuals of all living genera.
Eshel Ben-Jacob is the biophysicist who has linked Luca Turin’s works from microbes to humans via what is now known about RNA-mediated events and the conserved molecular mechanisms of protein folding, which are required to link DNA repair to ecological adaptations. See for example:
2003 Bacterial self-organization: co-enhancement of complexification and adaptability in a dynamic environment
2009 Learning from Bacteria about Natural Information Processing
2013 MicroRNA-based regulation of epithelial–hybrid–mesenchymal fate determination
2014 Modeling putative therapeutic implications of exosome exchange between tumor and immune cells
My comment: Evolutionary theorists fail to link RNA-mediated events to differences between healthy cell type differentiation and pathology.
2015 Distinct E-cadherin-based complexes regulate cell behaviour through miRNA processing or Src and p120 catenin activity
E-cadherin and p120 catenin (p120) are essential for epithelial homeostasis, but can also exert pro-tumorigenic activities.
My comment: Obviously, most people want to know how to prevent the pro-tumorigenic activities. This requires them to learn more about homeostasis.
2015 DNA methylation pathways and their crosstalk with histone methylation
Methylation of DNA and of histone 3 at Lys 9 (H3K9) are highly correlated with gene silencing in eukaryotes from fungi to humans. Both of these epigenetic marks need to be established at specific regions of the genome and then maintained at these sites through cell division. Protein structural domains that specifically recognize methylated DNA and methylated histones are key for targeting enzymes that catalyse these marks to appropriate genome sites. Genetic, genomic, structural and biochemical data reveal connections between these two epigenetic marks, and these domains mediate much of the crosstalk.
My comment: The fact that two epigenetic marks must be established and maintained may annoy evolutionary theorists who have touted their pseudoscientific nonsense about mutations and evolution — until they can provide experimental evidence of biologically-based cause and effect that suggests anything besides the nutrient-dependent de novo creation of genes is required to begin to link the epigenetic landscape to the physical landscape of DNA in organized genomes such as the organized genome of octopuses.
2015 The octopus genome and the evolution of cephalopod neural and morphological novelties
The sequencing of the octopus genome integrates everything currently known about how ecological variation becomes RNA-mediated ecological adaptation. The title of the article frames what is known in the context of evolved morphological and behavioral novelties. However, no experimental evidence of biologically-based cause and effect suggest that behaviors “evolve.”
The fact that serious scientists still use the language of evolutionary theorists continues to alter the course of history based on de Vries definition of “mutation” and the overall ignorance of theorists who suggest virus-perturbed protein folding, which is obviously linked from viral microRNAs to genomic entropy can be beneficial.
Think about this. Everything starts with the nutrient-dependent RNA-mediated de novo creation of genes and proceeds to link DNA activation to ecological adaptation via RNA-mediated amino acids substitutions that are fixed in organized genomes by the physiology of reproduction in all genera — whether or not you believe it.