An alternative mode of bacterial quorum sensing?

By: James V. Kohl | Published on: August 22, 2017

Out of the Books Aug 22, 2017

Schoolchildren in Turkey will no longer be learning about evolution, NPR reports.

Submitted by jvkohl_2055414

…learning about evolution is not the primary function of the decision, but rather to use it as a building block for students to learn more about their ecology.

Israeli Middle Schools will teach the ridiculous theory so that students can learn the difference between theories and models of biophysically constrained biologically-based cause and effect before they are admitted to universities, where they will learn about the virus-driven degradation of messenger RNA, which links mutations to all pathology.

Sir Patrick Bateson: Zoologists Should Not ‘Hog’ Upcoming Royal Society Evolution Meeting

I’m not sure we’re going to be talking about a completely new set of ideas, a lot have been around for a while. Frankly, I think some evolutionary biologists have not shed their neo-Darwinist clothing. There are some conservative-minded biologists who still think of the organism as being essentially passive, a view about which I am particularly concerned. However, the overall movement in biology is to integrate different disciplines making it a very lively area at the moment. The molecular biologists are talking to the ethologists, the ecologists to the physiologists, the population geneticists to the paleontologists, and so forth.

Howard Bloom, and others like him,  have failed humanity after learning about Eshel Ben-Jacob’s works. Bloom and many others have ignored the facts about natural information processing in bacteria, which links the sense of smell from quorum sensing to our visual perception of mass and energy in the context of the space-time continuum.

Learning from Bacteria about Natural Information Processing

Olfaction Warps Visual Time Perception

The RhlR quorum-sensing receptor controls Pseudomonas aeruginosa pathogenesis and biofilm development independently of its canonical homoserine lactone autoinducer

Quorum sensing (QS) is a process of bacterial cell-to-cell communication that relies on the production, detection, and response to extracellular signaling molecules called autoinducers [1]. QS allows groups of bacteria to synchronously alter behavior in response to changes in the population density and species composition of the surrounding bacterial community [2,3].

Quorum sensing is nutrient energy-dependent and pheromone controlled. It links electrons to ecosystems in all living genera via biophysical constraints on viral latency that link supercoiled DNA to the prevention of virus-driven degradation of messenger RNA, which all serious scientists have linked from mutations to all pathology.
Researchers find an alternative mode of bacterial quorum sensing

Quorum sensing is crucial for P. aeruginosa’s adaptability. The process regulates the development of biofilms, the three-dimensional structures formed by large bacterial communities that promote their ability to establish and maintain infections. “P. aeruginosa strains harboring mutations in the quorum-sensing machinery are attenuated for virulence, and thus, interfering with quorum sensing holds promise for the development of novel anti-microbial therapies,” said Bassler, the Squibb Professor in Molecular Biology at Princeton University and a Howard Hughes Medical Institute Investigator.

Ecological adaptation is energy-dependent. The energy attenuates the virulence, which links the virus-driven degradation of messenger RNA from mutations to all pathology.

P. aeruginosa possesses similar quorum-sensing machinery to other species of bacteria. For example, it produces an enzyme called RhlI that synthesizes an autoinducer molecule known as N-butanoyl-L-homoserine lactone, or C4-HSL. This molecule can then bind and activate a protein called RhlR that regulates the expression of multiple genes that P. aeruginosa needs to form a biofilm and/or infect a host.

The energy-dependent weekend resurrection of the bacterial flagellum in P. fluorescens was reported in the context of two pheromone-controlled amino acid substitutions as if the substitutions were mutations. Now, Bassler’s group reports that an autoinducer molecule may be the required link to energy-dependent pheromone-controlled biophysically constrained vial latency.
Evolutionary Rewiring


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